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Journal of Alzheimers Disease & Parkinsonism received 4334 citations as per Google Scholar report
Articles published in Journal of Alzheimers Disease & Parkinsonism have been cited by esteemed scholars and scientists all around the world. Journal of Alzheimers Disease & Parkinsonism has got h-index 25, which means every article in Journal of Alzheimers Disease & Parkinsonism has got 25 average citations.
Following are the list of articles that have cited the articles published in Journal of Alzheimers Disease & Parkinsonism.
| 2023 | 2022 | 2021 | 2020 | 2019 | 2018 | 2017 | 2016 | |
|---|---|---|---|---|---|---|---|---|
Total published articles |
26 | 33 | 60 | 35 | 45 | 44 | 107 | 89 |
Conference proceedings |
0 | 33 | 0 | 0 | 82 | 213 | 118 | 158 |
Citations received as per Google Scholar, other indexing platforms and portals |
123 | 389 | 489 | 425 | 374 | 263 | 237 | 253 |
| Journal total citations count | 4334 |
| Journal impact factor | 2.04 |
| Journal 5 years impact factor | 3.14 |
| Journal cite score | 10.01 |
| Journal h-index | 25 |
| Journal h-index since 2019 | 22 |
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Cannabinoid CB 2 Receptor Modulation by the Transcription Factor NRF2 is Specific in Microglial Cells |
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Role of MSK1 in the induction of NF-?B by the chemokine CX3CL1 in microglial cells |
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Transcription factor NRF2 as a promising therapeutic target for Alzheimer's disease |
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CX3CR1-deficient microglia shows impaired signalling of the transcription factor NRF2: Implications in tauopathies |
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Optimization of vinyl sulfone derivatives as potent nuclear factor erythroid 2-related factor 2 (Nrf2) activators for Parkinson's disease therapy |
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Basic concepts on the role of nuclear factor erythroid-derived 2-like 2 (Nrf2) in age-related diseases |
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Cx3cr1?deficiency exacerbates alpha?synuclein?A53T induced neuroinflammation and neurodegeneration in a mouse model of Parkinson's disease |
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Nrf2/ARE pathway modulation by dietary energy regulation in neurological disorders |
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The anti-inflammatory and anti-oxidant mechanisms of the Keap1/Nrf2/ARE signaling pathway in chronic diseases |
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TFEB protein expression is reduced in aged brains and its overexpression mitigates senescence-associated biomarkers and memory deficits in mice |
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A novel transgenic zebrafish line allows for in vivo quantification of autophagic activity in neurons |
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Transgenic expression of a ratiometric autophagy probe specifically in neurons enables the interrogation of brain autophagy in vivo |
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DNAzyme-Based Lithium-Selective Imaging Reveals Higher Lithium Accumulation in Bipolar Disorder Patient-Derived Neurons |
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Clinical and imaging correlates of Impaired cerebral vasoreactivity in patients with Parkinson's disease |
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Cerebral Vasoreactivity Changes Over Time in Patients With Different Clinical Manifestations of Cerebral Small Vessel Disease |
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Ultrasonographic Changes in Brain Hemodynamics in Patients with Parkinson's Disease and Risk Factors for Cerebrovascular Disease: A Pilot Study |
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With great interest we read the article recently published by Dadar and colleagues1 in Movement Disorders Clinical Practice. They report that in patients with early untreated de novo Parkinsonâs disease (PD), the interplay between dysautonomia and the burden of white matter hyperintensities (WMH) results in pronounced and accelerated cognitive decline. Although their results are of critical significance to help understand pathophysiological changes underlying cognitive decline and to address dysautonomia as a potential therapeutic target, we consider that there is 1 situation that has remained understudied in PD: cerebral vasomotor reactivity (CVR). |
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The renaissance of heart rate variability as a predictor of cognitive functioning |
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Potential clinical utility of multiple system atrophy biomarkers |
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Lipid Metabolism in Neurocognitive Disorders: Current Update |
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