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ISSN: 2376-127X

Journal of Pregnancy and Child Health
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  • Research Article   
  • J Preg Child Health,
  • DOI: 10.4172/2376-127X.1000380

Fetal Movement in Actocardiogram and Prevention of Cerebral Palsy with Hypoxia Index

Kazuo Maeda*
Department of Obstetrics and Gynaecology, Tottori University Medical School, , Japan
*Corresponding Author : Kazuo Maeda, Department of Obstetrics and Gynaecology, Tottori University Medical School, Yonago, Japan, Tel: +81859226856, Email: [email protected]

Received Date: May 17, 2018 / Accepted Date: Jun 05, 2018 / Published Date: Jun 12, 2018

Abstract

Aim: To analyze the relation of fetal movement and heart rate.
Method: Fetal movement and heart rate (FHR) was analysed by actodardiogram. Novel hypoxia index was calculated by the sum of FHR deceleration duration and the lowest FHR.
Results: As FHR increasesd when the fetus moved, FHR was studied in the relation to fetal movements, e,g, benign sinusoidal FHR was evoked by periodic fetal respiratory movement. No FHR acceleration was found in fetal hiccupping due to no formation of fetal movement burst. Hypoxic loss of FHR acceleration and variability were caused by no response of damaged fetal brain to fetal movement.
Discussion: The fetus should be cured from cerebral palsy by early delivery before the loss of variability, where the hypoxia index was 25 or more in the loss of variability followed by cerebral palsy, while normal variability and no cerebral palsy was preceded by 24 or less hypoxia index. Thus, the HI should be 24 or less to prevent cerebral palsy.
Conclusion: FHR changes, which had not solved by CTG, was solved by the application of fetal movement. Novel hypoxia index is usefull for the prevention of cerebral palsy by the 24 or less of hypoxia index.

Keywords: Fetus; Heart Rate; Brain Damage; Cerebral Palsy; Hypoxia Index; Ultrasound; Doppler Signal

Citation: Maeda K (2018) Fetal Movement in Actocardiogram and Prevention of Cerebral Palsy with Hypoxia Index. J Preg Child Health 5: 380. Doi: 10.4172/2376-127X.1000380

Copyright: © 2018 Maeda K. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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