Abstract

Genomic instability is a hallmark of cancer progression and plays a crucial role in cervical cancer development. The accumulation of genetic mutations, chromosomal aberrations, and DNA repair deficiencies contribute to tumor initiation, progression, and resistance to therapy. This article explores the impact of genomic instability on cervical cancer progression, focusing on the mechanisms driving instability, its clinical implications, and potential therapeutic targets. Understanding these factors is essential for improving diagnostic strategies, personalized treatment approaches, and patient outcomes.