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A Shield against Rift Valley Fever: Monoclonal Antibody Strikes the Vulnerable Site | OMICS International| Abstract
ISSN: 2167-7719

Air & Water Borne Diseases
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  • Review Article   
  • Air Water Borne Dis 12: 190.,
  • DOI: 10.4172/2167-7719.1000190

A Shield against Rift Valley Fever: Monoclonal Antibody Strikes the Vulnerable Site

Christian Schindler*
Department of Immunology and Microbiology, The Scripps Research Institute, La Jolla, USA
*Corresponding Author : Christian Schindler, Department of Immunology and Microbiology, The Scripps Research Institute, La Jolla, USA, Email: Schindler334@gmail.com

Received Date: Jun 02, 2023 / Published Date: Jun 30, 2023

Abstract

Rift Valley Fever (RVF) is a severe viral disease with significant public health and agricultural implications. In this article, we highlight a groundbreaking discovery—a monoclonal antibody that specifically targets a vulnerable site on the surface of the Rift Valley Fever virus. Through innovative research methods, scientists identified this vulnerable site crucial for viral entry and replication. The monoclonal antibody, designed to bind tightly to the site, acts as a shield, preventing viral attachment to host cells and neutralizing the virus. This breakthrough has immense potential for both therapeutic and preventive interventions against RVF. However, further research, clinical trials, and large-scale production are necessary to fully harness the potential of this monoclonal antibody. Nevertheless, this discovery represents a significant advancement in our ability to combat Rift Valley Fever and offers hope for improved disease control strategies.

Citation: Schindler C (2023) A Shield against Rift Valley Fever: Monoclonal Antibody Strikes the Vulnerable Site. Air Water Borne Dis 12: 190. Doi: 10.4172/2167-7719.1000190

Copyright: © 2023 Schindler C. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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