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Arrhythmia: Open Access
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Case Report

Impact of Nocturnal Oxygen Therapy for Central Sleep Apnea Syndrome on Nocturnal Premature Ventricular Contractions

Takeshi Tomita1*, Takeo Higashikata1, Ei-ichiro Mawatari1, Tsunemi Koji1, Harumi Sasaki2, KojiYoshie3, Yasutaka Oguchi3, Takahiro Takeuchi3, Ayako Okada3 and Uichi Ikeda3
1Department of Cardiovascular Medicine, North Alps Medical Center Azumi Hospital3207-1 Ikeda Kita-Azumi, Nagano, Japan
2Clinical laboratory, North Alps Medical Center Azumi Hospital 3207-1 Ikeda Kita-Azumi, Nagano, Japan
3Department of Cardiovascular Medicine, Shinshu University School of Medicine3-1-1 Asahi Matsumoto, Nagano, Japan
Corresponding Author : Takeshi Tomita
Department of Cardiovascular Medicine
North Alps Medical Center Azumi Hospital 3-1-1 Asahi Matsumoto Nagano, Japan
Tel: +81-263-37-348
E-mail: [email protected]
Received: September 20, 2015; Accepted: October 15, 2015; Published: October 23, 2015
Citation: Tomita T, Higashikata T, Mawatari E, Koji E, Sasaki H, et al. (2015) Impact of Nocturnal Oxygen Therapy for Central Sleep Apnea Syndrome on Nocturnal Premature Ventricular Contractions. Arrhythm Open Access 1:101. doi:10.4172/atoa.1000101
Copyright: © 2015 Tomita T, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
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Abstract

We report here a case of central sleep apnea syndrome associated with frequent nocturnal premature ventricular contractions (PVCs) that were successfully eliminated using nocturnal oxygen therapy. He was admitted to our hospital for chest discomfort during the night. The symptoms frequently woke him up during sleep. His electrocardiogram (ECG) showed sinus rhythm accompanied by PVCs. The 24 hour ambulatory ECG detected frequent PVCs during the night (more than 1000 beats/hour). Additionally, polysomnography demonstrated severe central sleep apnea syndrome, the so-called Cheyne-Stokes respirations (apnea-hypopnea index 48/hour). He received oxygen at a rate of 2 liter/min only during sleep. After nocturnal nasal oxygen therapy, the PVCs became significantly fewer and his symptoms disappeared. This case report suggests the possibility of an impact of oxygen inhalation during sleep in patients with central SAS on the prevention of ventricular arrhythmias during both the daytime and night. However, further well-established randomized, controlled studies are needed to confirm these effects.

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