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Vascular and Cardiac Valve Calcification in Chronic Kidney Disease | OMICS International| Abstract

Atherosclerosis: Open Access
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  • Review Article   
  • Atheroscler Open Access 2016, Vol 1(2): 104

Vascular and Cardiac Valve Calcification in Chronic Kidney Disease

Lirong Hao*, Xueying Chang, Yuting Fu and Zhangxiu He
Department of Nephropathy and Hemodialysis, , First Affiliated Hospital of Harbin Medical University, China
*Corresponding Author : Lirong Hao, Department of Nephropathy and Hemodialysis, First Affiliated Hospital of Harbin Medical University, Harbin, China, Email: [email protected]

Received Date: Sep 19, 2016 / Accepted Date: Nov 01, 2016 / Published Date: Nov 08, 2016

Abstract

Vascular calcification (VC) and cardiac valve calcification (CVC) are the important causes to increase the risk of cardiovascular events in terms of chronic kidney disease (CKD) patients. Once VC and CVC considered a passive form of dead or dying cells, it has now emerged as a pathology results from an active and highly regulated cellular process. Recently, mechanisms of VC have been further elucidated and many of the pathways involved could be amplified in CKD patients. In particular, FGF-23/Klotho axis, Wnt pathways, PI3K/Akt signaling, P38MAPK signaling pathway, and microRNAs have been shown to be impaired among patients with CKD and could play a role during vascular calcification. Furthermore, risks for CVC in CKD patients and molecular mechanisms related to it were verified by several researchers. The scope of the present review is to summarize the risk factors and pathophysiological mechanisms potentially involved in the link between CKD and the progression of VC and CVC.

Keywords: Chronic kidney disease; Vascular calcification; Cardiac valve calcification; Mechanisms

Citation: Hao L, Chang X, Fu Y, He Z (2016) Vascular and Cardiac Valve Calcification in Chronic Kidney Disease. Atheroscler open access 1: 104.

Copyright: © 2016 Hao L, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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