alexa Smoking, Periodontitis and Vascular Disease -Collaboration Study with Dentists and Vascular Surgeons | OMICS International | Abstract
ISSN: 2376-032X

JBR Journal of Interdisciplinary Medicine and Dental Science
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Review Article

Smoking, Periodontitis and Vascular Disease -Collaboration Study with Dentists and Vascular Surgeons

Takehisa Iwai1* and Makoto Umeda2

1Tsukuba Vascular Center, and Buerger Disease Research Institute, Tatsuzawa, Moriya city, Japan

2Department of Periodontology, Osaka Dental University, Japan

Corresponding Author:
Takehisa Iwai
Director and Head Tsukuba Vascular Center
and Buerger Disease Research Institute
Tatsuzawa, Moriya city, Japan
Tel: +81(297)45-47-9955
Fax: +81(297)45-4541
E-mail: [email protected]

Received Date: December 26, 2013; Accepted Date: February 05, 2014; Published Date: February 10, 2014

Citation: Iwai T, Umeda M (2014) Smoking, Periodontitis and Vascular Disease -Collaboration Study with Dentists and Vascular Surgeons. J Interdiscipl Med Dent Sci 2:113. doi: 10.4172/2376-032X.1000113

Copyright: © 2014 Iwai T, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited


Weak oral bacteria such as periodontal bacteria have been observed in various arterial and venous lesions with epidemiological data reported prior to the discovery of bacterial invasion into vessels. Rich lymph vessels easily bring the bacteria from the mouth to the neck and the venous angle, which is directly open to the blood vessels. Periodontal bacteria travel within platelets. Periodontal bacteria, especially P. gingivalis aggregates platelets and forms thrombus. At the same time, secretions such as serotonin, various cytokines, and adhesion factors also appear in the blood. The characteristic of the arterial lesions are dependent on the age of the patient and the condition of the endothelial cells. In young patients, infectious incidents occur due to embolic mechanisms in Buerger disease or adhesion to the superficial veins valves in varicose veins. In aged patients, incidents result in adhesion in the proximal aorta, coronary arteries or large arteries. The hypotheses here unify the evidence or vessel lesion development and explain possible discrepancy between vascular diseases. We were able to emphasize the collaboration study in this study with dentists and vascular surgeons.


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