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Multitarget Therapies In The Context Of The Aged Associated Oxidative Stress Induced Cellular And Subcellular And Vascular Hypoperfusion And Mitochondrial DNA Deletion During The Development And Maturation Of Alzheimer Disease: Past, Present And Future | 105586
ISSN: 2161-0460

Journal of Alzheimers Disease & Parkinsonism
Open Access

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Multitarget therapies in the context of the aged associated oxidative stress induced cellular and subcellular and vascular hypoperfusion and mitochondrial DNA deletion during the development and maturation of Alzheimer disease: past, present and future

11th International Conference on Vascular Dementia

Gjumrakch Aliev

“GALLY” International Biomedical Research Institute Inc., USAUniversity of Atlanta, Atlanta, USARussian Academy of Sciences, Russia

Posters & Accepted Abstracts: J Alzheimers Dis Parkinsonism

DOI: 10.4172/2161-0460-C1-061

Stroke and arteriosclerosis with neurological consequences such as Alzheimer disease (AD) are two leading causes of ageassociated disability, dementia, and death. AD is now the sixth-leading cause of death in the United States. In the US, AD is estimated to affect 4 million people (rising steeply from <1% of the population aged 65 to 40% of those aged 90) and costs $600 billion per year, which is equivalent to the total cost of stroke, heart disease, and cancer combined. Overall, there are no effective strategies for determining and controlling this devastating disease. Because AD is a multifactorial pathology and the development of new multitarget neuroprotective drugs is promising and attractive.