Reach Us +1-218-451-2974


Neuroinflammation & Alzheimer′s Disease | 79035
ISSN: 2161-0460

Journal of Alzheimers Disease & Parkinsonism
Open Access

Like us on:

Our Group organises 3000+ Global Conferenceseries Events every year across USA, Europe & Asia with support from 1000 more scientific Societies and Publishes 700+ Open Access Journals which contains over 50000 eminent personalities, reputed scientists as editorial board members.

Open Access Journals gaining more Readers and Citations
700 Journals and 15,000,000 Readers Each Journal is getting 25,000+ Readers

This Readership is 10 times more when compared to other Subscription Journals (Source: Google Analytics)
Recommended Conferences
Google scholar citation report
Citations : 2275

Journal of Alzheimers Disease & Parkinsonism received 2275 citations as per google scholar report

Journal of Alzheimers Disease & Parkinsonism peer review process verified at publons
Indexed In
  • Index Copernicus
  • Google Scholar
  • Sherpa Romeo
  • Open J Gate
  • Genamics JournalSeek
  • Academic Keys
  • JournalTOCs
  • China National Knowledge Infrastructure (CNKI)
  • Electronic Journals Library
  • RefSeek
  • Hamdard University
  • OCLC- WorldCat
  • SWB online catalog
  • Virtual Library of Biology (vifabio)
  • Publons
  • Geneva Foundation for Medical Education and Research
  • Euro Pub
Share This Page

Neuroinflammation & Alzheimer′s disease

9th International Conference on Alzheimers Disease & Dementia

Mai Mwafy, Paul Morgan, Shelley Allen and Patrick Kehoe

Bristol School of Medicine, UK Cardiff University, UK

Posters & Accepted Abstracts: J Alzheimers Dis Parkinsonism

DOI: 10.4172/2161-0460-C1-034

Neuroinflammation is a common feature of Alzheimer’s disease pathology, which is characterized by the presence of reactive astrocytes and activation of the microglia (the brain’s resident macrophages), as well as increased expression of pro-inflammatory cytokines & complement system activation. Amyloid beta protein accumulation in the brain of Alzheimer’s disease patients is the activator of the complement system and leads to glial cells activation and subsequent release of neurotoxic substances and free oxygen radicals. We are studying different aspects of neuroinflammation in a cohort of two groups “postmortem human brain tissue” of Alzheimer’s disease and age matched controls in different brain areas including frontal and temporal cortices to highlight the role of innate immunity in the disease and if it can be considered as potential targets for treating Alzheimer’s disease.

Mai Mwafy is currently a PhD student in University of Bristol, Dementia Research Group, also work in Infection & Immunity as a collaborator in Cardiff University, Mai has completed her Master’s Degree in Medical Microbiology & Immunology in Egypt, Faculty of Medicine Tanta University in 2013. Funded by: Newton- Mosharafa fund British Council in Egypt in collaboration with the Egyptian Government awarded 2015, and ARUK small grant awarded 2017.