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CMV Driven Immunosenescence and Alzheimers Disease
| Coad Thomas Dow* | |
| Mc Pherson Eye Research Institute University of Wisconsin-Madison, USA | |
| Corresponding Author : | Coad Thomas Dow Mc Pherson Eye Research Institute University of Wisconsin-Madison 2715 Damon Street Eau Claire Wisconsin, USA Tel: 7158348471 E-mail: ctdow@me.com |
| Received November 24, 2015; Accepted November 25, 2015; Published November 30, 2015 | |
| Citation: Dow CT (2015) CMV Driven Immunosenescence and Alzheimer’s Disease. J Neuroinfect Dis 6:195. doi:10.4172/2314-7326.1000195 | |
| Copyright: © 2015 Dow CT. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. | |
| Related article at Pubmed, Scholar Google | |
Abstract
Cytomegalovirus (CMV) is well known for producing severe life-threatening infection in immunocompromised hosts (HIV, organ transplant). CMV latently infects immune competent individuals as well and has been linked to Alzheimer’s disease. Nearly all elderly carry the CMV virus and the immune consequence of CMV can be overwhelming. This article reviews Alzheimer’s and its pathology, CMV and immune risk and how the immune dysfunction can lead to Alzheimer’s. Early risk detection with new biomarker imaging technology for Alzheimer’s along with antiviral drugs and immune support with telomerase activation may all mitigate the expected epidemic of Alzheimer’s disease.
