E-ISSN: 2314-7326
P-ISSN: 2314-7334

Journal of Neuroinfectious Diseases
Open Access

Our Group organises 3000+ Global Conferenceseries Events every year across USA, Europe & Asia with support from 1000 more scientific Societies and Publishes 700+ Open Access Journals which contains over 50000 eminent personalities, reputed scientists as editorial board members.

Open Access Journals gaining more Readers and Citations
700 Journals and 15,000,000 Readers Each Journal is getting 25,000+ Readers

This Readership is 10 times more when compared to other Subscription Journals (Source: Google Analytics)

Mini Review

CMV Driven Immunosenescence and Alzheimers Disease

Coad Thomas Dow*
Mc Pherson Eye Research Institute University of Wisconsin-Madison, USA
Corresponding Author : Coad Thomas Dow
Mc Pherson Eye Research Institute University of Wisconsin-Madison
2715 Damon Street
Eau Claire Wisconsin, USA
Tel: 7158348471
E-mail: ctdow@me.com
Received November 24, 2015; Accepted November 25, 2015; Published November 30, 2015
Citation: Dow CT (2015) CMV Driven Immunosenescence and Alzheimer’s Disease. J Neuroinfect Dis 6:195. doi:10.4172/2314-7326.1000195
Copyright: © 2015 Dow CT. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Related article at Pubmed, Scholar Google

Abstract

Cytomegalovirus (CMV) is well known for producing severe life-threatening infection in immunocompromised hosts (HIV, organ transplant). CMV latently infects immune competent individuals as well and has been linked to Alzheimer’s disease. Nearly all elderly carry the CMV virus and the immune consequence of CMV can be overwhelming. This article reviews Alzheimer’s and its pathology, CMV and immune risk and how the immune dysfunction can lead to Alzheimer’s. Early risk detection with new biomarker imaging technology for Alzheimer’s along with antiviral drugs and immune support with telomerase activation may all mitigate the expected epidemic of Alzheimer’s disease.

Keywords

Top