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Editor - Mohammad Moshahid Khan | The University of Iowa | 13279
E-ISSN: 2314-7326
P-ISSN: 2314-7334

Journal of Neuroinfectious Diseases
Open Access

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Mohammad Moshahid Khan

Mohammad Moshahid Khan
Mohammad Moshahid Khan
Research Scientist
Department of Neurology
University of Iowa Hospitals and Clinics
USA

Biography

Currently Dr. Khan is serving as Staff Scientist in the Department of Neurology at University of Iowa, Iowa, USA. He did his B.Sc. (Chemistry) in 2003, M.Sc. (Organic Chemistry) in 2005 from Aligarh Muslim University and Ph.D. (Toxicology) in 2009 from the department of Toxicology, Hamdard University, Delhi, India. After completion of his Ph.D. program,he has joined the Department of Internal medicine, University of Iowa as postdoctoral Research Associate. Here he focused his research on understanding the role of plasma glycoprotein von Willebrand factor (VWF), ADAMTS13 and EDA-fibronectin in animal model of stroke. After that he joined the department of Neurology as an Associate Scientist. Here his research investigates the molecular mechanisms that regulate inflammation in the central nervous system that are critical to our understanding of a broad spectrum of neurodegenerative diseases, including Parkinson’s disease, stroke, Alzheimer’s disease, multiple sclerosis and traumatic brain injury.He is the member of several international societies and enlisted in the Editorial board and reviewer list of the several journals of international repute.He has published almost 40 peer reviewed original research articles and almost 50 abstracts in National and International Neuroscience-related meeting and conferences. Dr. Khan research is committed to unraveling the mysteries of neurodegenerative diseases and to discovering new treatments for those incurable diseases.

Research Interest

My research uses genetic, cell biological and biochemical approaches to explore the pathogenesis of neurodegenerative diseases. We also investigate several discrete mechanisms involved in neuronal cell death including the role of proinflammatory protein, Glia maturation factor following neuronal death. Models of neurodegenration, their behavioural assessment and the effect of neuronal stem cell, protein specific siRNA and pharmacologically active candidates on the behavioral, neurochemical and histological recovery .

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